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Harnessing anti-tumor and tumor-tropism functions of macrophages via nanotechnology for tumor immunotherapy
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Recent advancements in drug delivery systems, particularly nanotechnology, have further enhanced the feasibility of melatonin-based ocular treatments by improving its solubility, stability, and permeability across ocular barriers (77, 78)
The RIPK1 inhibitor RIPA-56 significantly attenuates tubular epithelial cell necroptosis, reduces lipid deposition, and alleviates diabetic renal injury in STZ/HFD-treated db/db mice (84)
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